![]() The utility of mobile telephone-recorded videos as adjuncts to the diagnosis of seizures and paroxysmal events in children with suspected epileptic seizures 1 (2023)Ĭerebral palsy and its medicolegal implications in low- resource settings – the need to establish causality and revise criteria to implicate intrapartum hypoxia: A narrative review western-cape (accessed ).Īcute obstructive hydrocephalus in posterior reversible encephalopathy syndrome Hospital Level (Adults) Standard Treatment Guidelines and Essential Medicines List. National Department of Health, South Africa. Methylmercury: A potential environmental risk factor contributing to epileptogenesis. MR findings of Minamata disease – organic mercury poisoning. RopperAH,AdamsRD,VictorM,etal.AdamsandVictor’: McGraw-Hill, 2005. Gang battles over abandoned gold mines in South Africa. work_/keysectors/Pages/Mining.aspx#:~:text=The%20major%20gold%20and%20diamond,South%20 Africa’s%20total%20mineral%20production (accessed ). Burden of disease of mercury used in artisanal small-scale gold mining. Abandoned artisanal gold mines in the Brazilian Amazon: A legacy of mercury pollution. Mercury toxicity potential from artisanal and small-scale gold mines in Lebong Regency, Bengkulu Province. (accessed 14 February 2022).Īli M, Hery S, Putri SA. The impact of artisanal mining on rehabilitation efforts of abandoned mine shafts in Sutherland goldfield, South Africa. Mhlongo SE, Amponsah-Dacosta F, Muzerengi C, et al. Chronic neurological disease due to methylmercury poisoning. Percutaneous absorption of mercury vapor by man. Mercury toxicity and treatment: A review of the literature. Mercury hazards from gold mining to humans, plants, and animals. Methylmercury poisoning in Iraq: An interuniversity report. Tokyo: Government of Japan, 2002.īakir F, Damluji SF, Amin Zaki I. īerlinM,ZalupsRK,:Elsevier,2005.Įnvironmental Health Department, Ministry of the Environment, Japan. The toxicology of mercury and its compounds. A prompt diagnosis and the commencement of early chelation therapy have the potential to produce good outcomes. It is important to maintain a high index of suspicion for mercury poisoning, even in patients with atypical and unilateral or asymmetrical presentations. The neurological manifestations of mercury toxicity are typically symmetrical, whereas our two patients presented with markedly asymmetrical features. Patient 1 responded well to chelation therapy, but patient 2 refused admission and further medical treatment. ![]() Both patients had toxic mercury levels, with no other cause identified for their symptoms. Patient 2 had unilateral cerebellar ataxia. Patient 1 presented with focal seizures, an asymmetrical cerebellar syndrome and an acute encephalopathy. Here we describe the cases of two artisanal gold miners from western Johannesburg, South Africa, who presented with atypical neurological manifestations of mercury toxicity. Mercury is used in the gold mining process, and in artisanal or illicit gold mining, often without necessary protection. Previously described neurological manifestations of mercury toxicity are symmetrical, and include a pancerebellar syndrome, generalised seizures and encephalopathy. ![]() Mercury is a highly toxic heavy metal that may cause neurological, respiratory, gastrointestinal and dermatological illnesses. Mercury, Toxicity, Miners, Informal, Neurology, Ataxia, Encephalopathy, Seizures Abstract Division of Neurology, Department of Neurosciences, School of Clinical Medicine, Faculty of Health Sciences, University of the Witwatersrand, Johannesburg, South Africaĭepartment of Internal Medicine, School of Clinical Medicine, Faculty of Health Sciences, University of the Witwatersrand, Johannesburg, South Africa ![]()
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